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Alzheimer’s Disease? Information about Alzheimer’s disease
Although some types of memory loss a normal part of aging, the changes due to aging are not strong enough to interfere with the plane of the function. Many different diseases can be to dementia, but Alzheimer’s disease by far the most common cause of dementia in the United States and in most countries of the world.
Dementia is a brain disorder seriously affects a person’s ability to carry out daily activities. The most common form of dementia in older people is Alzheimer’s disease (AD), which comprises the first part of the brain that control thought, memory and language. Although scientists learning more every day, still do not now know what causes AD, and there is no cure.
There is no cure for Alzheimer’s and no way to slow the progression of the disease. For some people in the early or middle stages of AD, medication such as tacrine (Cognex) may alleviate some cognitive symptoms. Donepezil (Aricept), rivastigmine (Exelon) and galantamine (Reminyl), some symptoms are worse by keeping for a limited time. A fifth drug, memantine (Namenda), was recently approved for use in the United States. The combination of memantine with other.
The probability of Alzheimer’s disease increases substantially after the age of 70 and may affect about 50% of people over 85 years. However, the Alzheimer’s disease is not a normal part of aging and is not something that inevitably happens in later life. So many people live in over 100 years old and never develop Alzheimer’s disease.
AD drugs may be more effective than any single therapy. A controlled clinical trial showed that patients receiving donepezil plus memantine better cognition and other functions than patients receiving donepezil alone. It can also help other medications to control behavioral problems such as insomnia, restlessness, hiking, anxiety and depression.
The tracer called Pittsburgh Compound B, was during a PET scan in a woman with a diagnosis of Alzheimer’s disease 10 months before her death. The tracer showed plaque formation in the same areas of the brain where plaques at autopsy.